Evaluating the role of the 620W allele of protein tyrosine phosphatase PTPN22 in Crohn's disease and multiple sclerosis
Philip L. De Jager(Broad Institute), John D. Rioux(Institute for Molecular Medicine Finland), Alicja Waliszewska(Broad Institute), Gary Wild(Montreal General Hospital), Stephen Sawcer(University of Cambridge), Alastair Compston(University of Cambridge), Diane Langelier(Centre Hospitalier Universitaire de Sherbrooke), Lisa Farwell(Broad Institute), David A. Hafler(Yale University), Albert Cohen(Jewish General Hospital), Alain Bitton(McGill University Health Centre)
Cited by 39
Related Papers
Host–microbe interactions have shaped the genetic architecture of inflammatory bowel disease
|Nature|2012|4.8k
Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease
|Nature Genetics|2013|4.7k
A Genome-Wide Association Study Identifies <i>IL23R</i> as an Inflammatory Bowel Disease Gene
|Science|2006|3k
Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis
|Nature|2011|2.8k
Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease
|Nature Genetics|2008|2.6k