Upregulation of K <sub>2P</sub> 3.1 K <sup>+</sup> Current Causes Action Potential Shortening in Patients With Chronic Atrial FibrillationConstanze Schmidt, Dierk Thomas, Felix Wiedmann et al.|Circulation|2015Cited by 195
Role of ion channels in heart failure and channelopathiesAnn‐Kathrin Rahm, Dierk Thomas, Patrick A. Schweizer et al.|Biophysical Reviews|2018Cited by 68
TREK-1 (K2P2.1) K+ channels are suppressed in patients with atrial fibrillation and heart failure and provide therapeutic targets for rhythm controlPatrick Lugenbiel, Dierk Thomas, Fabian Wenz et al.|Basic Research in Cardiology|2016Cited by 48
Therapeutic targeting of two-pore-domain potassium (K2P) channels in the cardiovascular systemFelix Wiedmann, Dierk Thomas, Constanze Schmidt et al.|Clinical Science|2016Cited by 47
Cloning, functional characterization, and remodeling of K2P3.1 (TASK-1) potassium channels in a porcine model of atrial fibrillation and heart failureConstanze Schmidt, Dierk Thomas, Felix Wiedmann et al.|Heart Rhythm|2014Cited by 46