Upregulation of K <sub>2P</sub> 3.1 K <sup>+</sup> Current Causes Action Potential Shortening in Patients With Chronic Atrial FibrillationConstanze Schmidt, Dierk Thomas, Arjang Ruhparwar et al.|Circulation|2015Cited by 195
Therapeutic targeting of two-pore-domain potassium (K2P) channels in the cardiovascular systemFelix Wiedmann, Dierk Thomas, Constanze Schmidt et al.|Clinical Science|2016Cited by 47
Cloning, functional characterization, and remodeling of K2P3.1 (TASK-1) potassium channels in a porcine model of atrial fibrillation and heart failureConstanze Schmidt, Dierk Thomas, Felix Wiedmann et al.|Heart Rhythm|2014Cited by 46
Cardiac expression and atrial fibrillation-associated remodeling of K2P2.1 (TREK-1) K+ channels in a porcine modelConstanze Schmidt, Dierk Thomas, Felix Wiedmann et al.|Life Sciences|2013Cited by 45