Oncostatin M promotes cancer cell plasticity through cooperative STAT3-SMAD3 signalingDamian J. Junk, Mark W. Jackson|Oncogene|2017Cited by 134
HER2-positive breast cancer cells expressing elevated FAM83A are sensitive to FAM83A lossCourtney A. Bartel, Mark W. Jackson|PLoS ONE|2017Cited by 69
Oncostatin-M and OSM-Receptor Feed-Forward Activation of MAPK Induces Separable Stem-like and Mesenchymal ProgramsKelsey L. Polak, Mark W. Jackson|Molecular Cancer Research|2023Cited by 14
Toll-Like receptor 3-mediated interferon-β production is suppressed by oncostatin m and a broader epithelial-mesenchymal transition programNoah M. Chernosky, Mark W. Jackson|Breast Cancer Research|2024Cited by 1