Oncostatin-M and OSM-Receptor Feed-Forward Activation of MAPK Induces Separable Stem-like and Mesenchymal ProgramsKelsey L. Polak(Case Western Reserve University), Mark W. Jackson(Case Western Reserve University)Molecular Cancer ResearchJune 13, 202310.1158/1541-7786.mcr-22-0715Cited by 14SaveCiteExport RISWatch citationsRelated PapersOncostatin M promotes cancer cell plasticity through cooperative STAT3-SMAD3 signaling|Oncogene|2017|134HER2-positive breast cancer cells expressing elevated FAM83A are sensitive to FAM83A loss|PLoS ONE|2017|69Toll-Like receptor 3-mediated interferon-β production is suppressed by oncostatin m and a broader epithelial-mesenchymal transition program|Breast Cancer Research|2024|1