Astrocyte-Microglia Cross Talk through Complement Activation Modulates Amyloid Pathology in Mouse Models of Alzheimer's DiseaseHong Lian, Hui Zheng, Alexandra Litvinchuk et al.|Journal of Neuroscience|2016Cited by 600
Complement C3aR Inactivation Attenuates Tau Pathology and Reverses an Immune Network Deregulated in Tauopathy Models and Alzheimer’s DiseaseAlexandra Litvinchuk, Hui Zheng, Ying-Wooi Wan et al.|Neuron|2018Cited by 525
Practical considerations for choosing a mouse model of Alzheimer’s diseaseJoanna L. Jankowsky, Hui Zheng|Molecular Neurodegeneration|2017Cited by 455
Endothelial C3a receptor mediates vascular inflammation and blood-brain barrier permeability during agingNicholas E. Propson, Hui Zheng, Ethan R. Roy et al.|Journal of Clinical Investigation|2020Cited by 246
Complement C3aR depletion reverses HIF-1α–induced metabolic impairment and enhances microglial response to Aβ pathologyManasee Gedam, Hui Zheng|Journal of Clinical Investigation|2023Cited by 61