Complement C3aR depletion reverses HIF-1α–induced metabolic impairment and enhances microglial response to Aβ pathology
Manasee Gedam(Baylor College of Medicine), Hui Zheng(Hunan University)
Cited by 61
Related Papers
Astrocyte-Microglia Cross Talk through Complement Activation Modulates Amyloid Pathology in Mouse Models of Alzheimer's Disease
|Journal of Neuroscience|2016|600
Complement C3aR Inactivation Attenuates Tau Pathology and Reverses an Immune Network Deregulated in Tauopathy Models and Alzheimer’s Disease
|Neuron|2018|525
Practical considerations for choosing a mouse model of Alzheimer’s disease
|Molecular Neurodegeneration|2017|455
Endothelial C3a receptor mediates vascular inflammation and blood-brain barrier permeability during aging
|Journal of Clinical Investigation|2020|246