The Long Form of Fas Apoptotic Inhibitory Molecule Is Expressed Specifically in Neurons and Protects Them against Death Receptor-Triggered ApoptosisMiguel F. Segura, Joan X. Comella|Journal of Neuroscience|2007Cited by 77
TNFα induces survival through the FLIP-L-dependent activation of the MAPK/ERK pathwayFernando Marqués-Fernàndez, Joan X. Comella|Cell Death and Disease|2013Cited by 71
Amyloid-β reduces the expression of neuronal FAIM-L, thereby shifting the inflammatory response mediated by TNFα from neuronal protection to deathPaulina Carriba, Joan X. Comella|Cell Death and Disease|2015Cited by 40
FAIM-L Is an IAP-Binding Protein That Inhibits XIAP Ubiquitinylation and Protects from Fas-Induced ApoptosisRana S. Moubarak, Joan X. Comella|Journal of Neuroscience|2013Cited by 26
TNFα sensitizes neuroblastoma cells to FasL-, cisplatin- and etoposide-induced cell death by NF-κB-mediated expression of FasKoen M.O. Galenkamp, Joan X. Comella|Molecular Cancer|2015Cited by 24