TNFα induces survival through the FLIP-L-dependent activation of the MAPK/ERK pathway
Fernando Marqués-Fernàndez(Vall d'Hebron Institut de Recerca), Joan X. Comella(Universitat Autònoma de Barcelona)
Cited by 71
Related Papers
The Long Form of Fas Apoptotic Inhibitory Molecule Is Expressed Specifically in Neurons and Protects Them against Death Receptor-Triggered Apoptosis
|Journal of Neuroscience|2007|77
Amyloid-β reduces the expression of neuronal FAIM-L, thereby shifting the inflammatory response mediated by TNFα from neuronal protection to death
|Cell Death and Disease|2015|40
FAIM-L Is an IAP-Binding Protein That Inhibits XIAP Ubiquitinylation and Protects from Fas-Induced Apoptosis
|Journal of Neuroscience|2013|26
TNFα sensitizes neuroblastoma cells to FasL-, cisplatin- and etoposide-induced cell death by NF-κB-mediated expression of Fas
|Molecular Cancer|2015|24
The Death Receptor Antagonist FLIP-L Interacts with Trk and Is Necessary for Neurite Outgrowth Induced by Neurotrophins
|Journal of Neuroscience|2010|15