Host Genotype-Specific Therapies Can Optimize the Inflammatory Response to Mycobacterial InfectionsDavid M. Tobin, Lalita Ramakrishnan, Sungwhan F. Oh et al.|Cell|2012Cited by 572
Verapamil and its metabolite norverapamil inhibit the <i>Mycobacterium tuberculosis</i> MmpS5L5 efflux pump to increase bedaquiline activityAdam J. Fountain, Lalita Ramakrishnan|Proceedings of the National Academy of Sciences|2025Cited by 16
mTOR-regulated Mitochondrial Metabolism Limits Mycobacterium-induced CytotoxicityAntonio J. Pagán, Lalita Ramakrishnan|bioRxiv (Cold Spring Harbor Laboratory)|2022Cited by 5
Structural and functional analysis of the <i>Mycobacterium tuberculosis</i> MmpS5L5 efflux pump presages increased bedaquiline resistanceAdam J. Fountain, Lalita Ramakrishnan|Proceedings of the National Academy of Sciences|2025Cited by 4
The C terminus of the mycobacterium ESX-1 secretion system substrate ESAT-6 is required for phagosomal membrane damage and virulenceMorwan M. Osman, Lalita Ramakrishnan|bioRxiv (Cold Spring Harbor Laboratory)|2022Cited by 1