The C terminus of the mycobacterium ESX-1 secretion system substrate ESAT-6 is required for phagosomal membrane damage and virulence
Morwan M. Osman(MRC Laboratory of Molecular Biology), Lalita Ramakrishnan(MRC Laboratory of Molecular Biology)
Cited by 1
Related Papers
Verapamil and its metabolite norverapamil inhibit the <i>Mycobacterium tuberculosis</i> MmpS5L5 efflux pump to increase bedaquiline activity
|Proceedings of the National Academy of Sciences|2025|16
mTOR-regulated Mitochondrial Metabolism Limits Mycobacterium-induced Cytotoxicity
|bioRxiv (Cold Spring Harbor Laboratory)|2022|5
Structural and functional analysis of the <i>Mycobacterium tuberculosis</i> MmpS5L5 efflux pump presages increased bedaquiline resistance
|Proceedings of the National Academy of Sciences|2025|4
Verapamil and its metabolite norverapamil inhibit the <i>Mycobacterium tuberculosis</i> MmpS5L5 efflux pump to potentiate the activity bedaquiline and other antitubercular drugs
|bioRxiv (Cold Spring Harbor Laboratory)|2025|0