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Inactivation of the Tuberous Sclerosis Complex-1 and -2 Gene Products Occurs by Phosphoinositide 3-Kinase/Akt-dependent and -independent Phosphorylation of TuberinAndrew R. Tee, John Blenis|Journal of Biological Chemistry|2003Cited by 214
Endoplasmic reticulum stress and cell death in mTORC1‐overactive cells is induced by nelfinavir and enhanced by chloroquineC. E. Johnson, Andrew R. Tee|Molecular Oncology|2014Cited by 42
STAT3 and HIF1α Signaling Drives Oncogenic Cellular Phenotypes in Malignant Peripheral Nerve Sheath TumorsEllie Rad, Andrew R. Tee|Molecular Cancer Research|2015Cited by 33
Loss of tuberous sclerosis complex 2 sensitizes tumors to nelfinavir−bortezomib therapy to intensify endoplasmic reticulum stress-induced cell deathC. E. Johnson, Andrew R. Tee|Oncogene|2018Cited by 18