Inactivation of the Tuberous Sclerosis Complex-1 and -2 Gene Products Occurs by Phosphoinositide 3-Kinase/Akt-dependent and -independent Phosphorylation of Tuberin
Andrew R. Tee(Cardiff University), John Blenis(Harvard University)
Cited by 214
Related Papers
RAS/ERK Signaling Promotes Site-specific Ribosomal Protein S6 Phosphorylation via RSK and Stimulates Cap-dependent Translation
|Journal of Biological Chemistry|2007|752
STAT3 Serine Phosphorylation by ERK-Dependent and -Independent Pathways Negatively Modulates Its Tyrosine Phosphorylation
|Molecular and Cellular Biology|1997|622
Hypoxia-inducible Factor 1α Is Regulated by the Mammalian Target of Rapamycin (mTOR) via an mTOR Signaling Motif
|Journal of Biological Chemistry|2007|491
p90 Ribosomal S6 Kinase and p70 Ribosomal S6 Kinase Link Phosphorylation of the Eukaryotic Chaperonin Containing TCP-1 to Growth Factor, Insulin, and Nutrient Signaling
|Journal of Biological Chemistry|2009|90
Rap1-GTPases control mTORC1 activity by coordinating lysosome organization with amino acid availability
|Nature Communications|2020|81