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Common Defects of ABCG2, a High-Capacity Urate Exporter, Cause Gout: A Function-Based Genetic Analysis in a Japanese PopulationHirotaka Matsuo, Nariyoshi Shinomiya, Tappei Takada et al.|Science Translational Medicine|2009Cited by 434
Genome-wide association study of clinically defined gout identifies multiple risk loci and its association with clinical subtypesHirotaka Matsuo, Nariyoshi Shinomiya, Ken Yamamoto et al.|Annals of the Rheumatic Diseases|2015Cited by 183
GWAS of clinically defined gout and subtypes identifies multiple susceptibility loci that include urate transporter genesAkiyoshi Nakayama, Hirotaka Matsuo, Hirofumi Nakaoka et al.|Annals of the Rheumatic Diseases|2016Cited by 173
ABCG2 dysfunction causes hyperuricemia due to both renal urate underexcretion and renal urate overloadHirotaka Matsuo, Nariyoshi Shinomiya, Akiyoshi Nakayama et al.|Scientific Reports|2014Cited by 163