Genome-wide association study of clinically defined gout identifies multiple risk loci and its association with clinical subtypesHirotaka Matsuo, Nariyoshi Shinomiya, Seiko Shimizu et al.|Annals of the Rheumatic Diseases|2015Cited by 183
GWAS of clinically defined gout and subtypes identifies multiple susceptibility loci that include urate transporter genesAkiyoshi Nakayama, Hirotaka Matsuo, Hirofumi Nakaoka et al.|Annals of the Rheumatic Diseases|2016Cited by 173
ABCG2 dysfunction causes hyperuricemia due to both renal urate underexcretion and renal urate overloadHirotaka Matsuo, Nariyoshi Shinomiya, Akiyoshi Nakayama et al.|Scientific Reports|2014Cited by 163
Genome-wide association study revealed novel loci which aggravate asymptomatic hyperuricaemia into goutYusuke Kawamura, Hirofumi Nakaoka, Yukinori Okada et al.|Annals of the Rheumatic Diseases|2019Cited by 103
Common dysfunctional variants of ABCG2 have stronger impact on hyperuricemia progression than typical environmental risk factorsAkiyoshi Nakayama, Nariyoshi Shinomiya, Hirotaka Matsuo et al.|Scientific Reports|2014Cited by 96