Cell Signaling Technology

WDR5 (D3X5B) Rabbit Monoclonal Antibody

#13073 · $161

100
Citing papers
5.1k
Combined citations
Supplier page Find this product

Research areas

Endoplasmic Reticulum Stress and Disease23Autophagy in Disease and Therapy8Cytomegalovirus and herpesvirus research8Cancer-related Molecular Pathways7DNA Repair Mechanisms5Herpesvirus Infections and Treatments5Cancer, Hypoxia, and Metabolism5Viral-associated cancers and disorders5

Papers citing this product

1
Impaired autophagic flux is associated with increased endoplasmic reticulum stress during the development of NAFLD
Cell Death and Disease·2014·622 cit.·DOI
2
IL-6 and IL-10 Anti-Inflammatory Activity Links Exercise to Hypothalamic Insulin and Leptin Sensitivity through IKKβ and ER Stress Inhibition
PLoS Biology·2010·324 cit.·DOI
3
Palmitate induces ER calcium depletion and apoptosis in mouse podocytes subsequent to mitochondrial oxidative stress
Cell Death and Disease·2015·200 cit.·DOI
4
Fluid shear triggers microvilli formation via mechanosensitive activation of TRPV6
Nature Communications·2015·183 cit.·DOI
5
Neuregulin-ErbB Signaling Promotes Microglial Proliferation and Chemotaxis Contributing to Microgliosis and Pain after Peripheral Nerve Injury
Journal of Neuroscience·2010·173 cit.·DOI
6
Role of Pro-oncogenic Protein Disulfide Isomerase (PDI) Family Member Anterior Gradient 2 (AGR2) in the Control of Endoplasmic Reticulum Homeostasis
Journal of Biological Chemistry·2011·172 cit.·DOI
7
Macrophage metabolic reprogramming aggravates aortic dissection through the HIF1α-ADAM17 pathway✰
EBioMedicine·2019·134 cit.·DOI
8
Cocaine-mediated induction of microglial activation involves the ER stress-TLR2 axis
Journal of Neuroinflammation·2016·119 cit.·DOI
9
An Epilepsy-Associated KCNT1 Mutation Enhances Excitability of Human iPSC-Derived Neurons by Increasing Slack K<sub>Na</sub> Currents
Journal of Neuroscience·2019·113 cit.·DOI
10
Osteoclasts protect bone blood vessels against senescence through the angiogenin/plexin-B2 axis
Nature Communications·2021·109 cit.·DOI

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