An Epilepsy-Associated KCNT1 Mutation Enhances Excitability of Human iPSC-Derived Neurons by Increasing Slack K<sub>Na</sub> CurrentsImran H. Quraishi(Yale University), Leonard K. Kaczmarek(Yale University)Journal of NeuroscienceJuly 26, 201910.1523/jneurosci.1628-18.2019Cited by 113SaveCiteExport RISWatch citationsRelated PapersLoss of Kv3.1 Tonotopicity and Alterations in cAMP Response Element-Binding Protein Signaling in Central Auditory Neurons of Hearing Impaired Mice|Journal of Neuroscience|2004|98Cerebellar Kv3.3 potassium channels activate TANK-binding kinase 1 to regulate trafficking of the cell survival protein Hax-1|Nature Communications|2021|28