Cell Signaling Technology

Phospho-eIF2 alpha (Ser51) (D9G8) Rabbit Monoclonal Antibody

#3398 · western blot · $1,791,070

65
Evidence Score · Well-Established
Used by 464 independent labs across 536 papers. How we score this →
100
Citing papers
16.7k
Combined citations
Supplier page Find this product

Research areas

Endoplasmic Reticulum Stress and Disease20Pancreatic function and diabetes10Autophagy in Disease and Therapy9Cancer, Hypoxia, and Metabolism6PI3K/AKT/mTOR signaling in cancer6RNA regulation and disease6MicroRNA in disease regulation5Alzheimer's disease research and treatments5

Papers citing this product

1
Distinct populations of inflammatory fibroblasts and myofibroblasts in pancreatic cancer
The Journal of Experimental Medicine·2017·2.4k cit.·DOI
2
Endoplasmic reticulum stress contributes to beta cell apoptosis in type 2 diabetes
Diabetologia·2007·819 cit.·DOI
3
<i>FBW7</i> mutations in leukemic cells mediate NOTCH pathway activation and resistance to γ-secretase inhibitors
The Journal of Experimental Medicine·2007·660 cit.·DOI
4
Disruption of endoplasmic reticulum structure and integrity in lipotoxic cell death
Journal of Lipid Research·2006·566 cit.·DOI
5
Palmitate Induces Insulin Resistance in H4IIEC3 Hepatocytes through Reactive Oxygen Species Produced by Mitochondria
Journal of Biological Chemistry·2009·428 cit.·DOI
6
Rheb Binding to Mammalian Target of Rapamycin (mTOR) Is Regulated by Amino Acid Sufficiency
Journal of Biological Chemistry·2005·346 cit.·DOI
7
Akt Signals through the Mammalian Target of Rapamycin Pathway to Regulate CNS Myelination
Journal of Neuroscience·2009·321 cit.·DOI
8
Axonal Regeneration and Lack of Astrocytic Gliosis in EphA4-Deficient Mice
Journal of Neuroscience·2004·309 cit.·DOI
9
Leukocyte Common Antigen-Related Phosphatase Is a Functional Receptor for Chondroitin Sulfate Proteoglycan Axon Growth Inhibitors
Journal of Neuroscience·2011·307 cit.·DOI
10
Serine synthesis pathway inhibition cooperates with dietary serine and glycine limitation for cancer therapy
Nature Communications·2021·301 cit.·DOI

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