Cell Signaling Technology

Phospho-eIF2 alpha (Ser51) (119A11) Rabbit Monoclonal Antibody

#3597 · $169

100
Citing papers
14.7k
Combined citations
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Research areas

Bone Metabolism and Diseases23Bone health and treatments15Endoplasmic Reticulum Stress and Disease12NF-κB Signaling Pathways12Autophagy in Disease and Therapy9Cytokine Signaling Pathways and Interactions7Acute Myeloid Leukemia Research6Chronic Myeloid Leukemia Treatments6

Papers citing this product

1
AC220 is a uniquely potent and selective inhibitor of FLT3 for the treatment of acute myeloid leukemia (AML)
Blood·2009·641 cit.·DOI
2
Essential Role of p38 Mitogen-activated Protein Kinase in Cathepsin K Gene Expression during Osteoclastogenesis through Association of NFATc1 and PU.1
Journal of Biological Chemistry·2004·443 cit.·DOI
3
Microtubules Facilitate Autophagosome Formation and Fusion of Autophagosomes with Endosomes
Traffic·2005·420 cit.·DOI
4
Critical roles of c-Jun signaling in regulation of NFAT family and RANKL-regulated osteoclast differentiation
Journal of Clinical Investigation·2004·417 cit.·DOI
5
Estrogen Decreases Osteoclast Formation by Down-regulating Receptor Activator of NF-κB Ligand (RANKL)-induced JNK Activation
Journal of Biological Chemistry·2001·355 cit.·DOI
6
Probiotics may delay the progression of nonalcoholic fatty liver disease by restoring the gut microbiota structure and improving intestinal endotoxemia
Scientific Reports·2017·306 cit.·DOI
7
Exosomes derived from hypoxic epithelial ovarian cancer deliver microRNA-940 to induce macrophage M2 polarization
Oncology Reports·2017·299 cit.·DOI
8
Activation of the Akt-NF-κB Pathway by Subtilase Cytotoxin through the ATF6 Branch of the Unfolded Protein Response
The Journal of Immunology·2009·283 cit.·DOI
9
Post-translational Modifications of Three Members of the Human MAP1LC3 Family and Detection of a Novel Type of Modification for MAP1LC3B
Journal of Biological Chemistry·2003·271 cit.·DOI
10
The SARS Coronavirus 3a Protein Causes Endoplasmic Reticulum Stress and Induces Ligand-Independent Downregulation of the Type 1 Interferon Receptor
PLoS ONE·2009·271 cit.·DOI

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