Cell Signaling Technology

Phospho-mTOR (Ser2448) Antibody

#2971 · $362,852

71
Evidence Score · Well-Established
Used by 659 independent labs across 706 papers. How we score this →
100
Citing papers
18.2k
Combined citations
Supplier page Find this product

Research areas

Autophagy in Disease and Therapy14PI3K/AKT/mTOR signaling in cancer7MicroRNA in disease regulation7RNA modifications and cancer6RNA Research and Splicing6Nerve injury and regeneration5Cancer-related molecular mechanisms research5Endoplasmic Reticulum Stress and Disease5

Papers citing this product

1
Trehalose, a Novel mTOR-independent Autophagy Enhancer, Accelerates the Clearance of Mutant Huntingtin and α-Synuclein
Journal of Biological Chemistry·2006·1.1k cit.·DOI
2
Definition of a novel growth factor-dependent signal cascade for the suppression of bile acid biosynthesis
Genes & Development·2003·681 cit.·DOI
3
Microbiota-derived short-chain fatty acids promote Th1 cell IL-10 production to maintain intestinal homeostasis
Nature Communications·2018·656 cit.·DOI
4
Impaired autophagic flux is associated with increased endoplasmic reticulum stress during the development of NAFLD
Cell Death and Disease·2014·622 cit.·DOI
5
P2X4-Receptor-Mediated Synthesis and Release of Brain-Derived Neurotrophic Factor in Microglia Is Dependent on Calcium and p38-Mitogen-Activated Protein Kinase Activation
Journal of Neuroscience·2009·463 cit.·DOI
6
The experimental power of FR900359 to study Gq-regulated biological processes
Nature Communications·2015·371 cit.·DOI
7
Predicting clinical response to anticancer drugs using an ex vivo platform that captures tumour heterogeneity
Nature Communications·2015·307 cit.·DOI
8
Fin56-induced ferroptosis is supported by autophagy-mediated GPX4 degradation and functions synergistically with mTOR inhibition to kill bladder cancer cells
Cell Death and Disease·2021·297 cit.·DOI
9
Deregulated MYC expression induces dependence upon AMPK-related kinase 5
Nature·2012·278 cit.·DOI
10
WNT signaling drives cholangiocarcinoma growth and can be pharmacologically inhibited
Journal of Clinical Investigation·2015·274 cit.·DOI

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