Cell Signaling Technology

Phospho-mTOR (Ser2448) (49F9) Rabbit Monoclonal Antibody

#2976 · $176

100
Citing papers
13k
Combined citations
Supplier page Find this product

Research areas

Cell death mechanisms and regulation11PI3K/AKT/mTOR signaling in cancer9interferon and immune responses8Chronic Lymphocytic Leukemia Research7Wnt/β-catenin signaling in development and cancer7Ubiquitin and proteasome pathways7Hippo pathway signaling and YAP/TAZ6Inflammasome and immune disorders6

Papers citing this product

1
EGFR-TKI resistance due to BIM polymorphism can be circumvented in combination with HDAC inhibition
Institutional Repositories DataBase (IRDB)·2014·741 cit.
2
FADD and Caspase-8 Mediate Priming and Activation of the Canonical and Noncanonical Nlrp3 Inflammasomes
The Journal of Immunology·2014·548 cit.·DOI
3
AIM2 and NLRP3 inflammasomes activate both apoptotic and pyroptotic death pathways via ASC
Cell Death and Differentiation·2013·495 cit.·DOI
4
The SWI/SNF chromatin remodelling complex is required for maintenance of lineage specific enhancers
Nature Communications·2017·436 cit.·DOI
5
Caspase-8 Serves Both Apoptotic and Nonapoptotic Roles
The Journal of Immunology·2004·377 cit.·DOI
6
Characterization of the Limbal Epithelial Stem Cell Niche: Novel Imaging Techniques Permit In Vivo Observation and Targeted Biopsy of Limbal Epithelial Stem Cells
Stem Cells·2007·310 cit.·DOI
7
Nurselike cells express BAFF and APRIL, which can promote survival of chronic lymphocytic leukemia cells via a paracrine pathway distinct from that of SDF-1α
Blood·2005·300 cit.·DOI
8
1,25(OH)2Vitamin D3 Stimulates Myogenic Differentiation by Inhibiting Cell Proliferation and Modulating the Expression of Promyogenic Growth Factors and Myostatin in C2C12 Skeletal Muscle Cells
Endocrinology·2011·258 cit.·DOI
9
Evidence That Ser87 of BimEL Is Phosphorylated by Akt and Regulates BimEL Apoptotic Function
Journal of Biological Chemistry·2005·230 cit.·DOI
10
STING inhibits the reactivation of dormant metastasis in lung adenocarcinoma
Nature·2023·225 cit.·DOI

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