Cell Signaling Technology

FLT3 (8F2) Rabbit Monoclonal Antibody

#3462 · $173,351

62

Evidence Score · Well-Established

Used by 130 independent labs across 142 papers. How we score this →

100

Citing papers

12.5k

Combined citations

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Research areas

Cancer Immunotherapy and Biomarkers12 Immune Cell Function and Interaction8 Alzheimer's disease research and treatments7 Immunotherapy and Immune Responses6 Autophagy in Disease and Therapy6 Neuroscience and Neuropharmacology Research6 interferon and immune responses5 Immune Response and Inflammation5

Papers citing this product

1

PROTAC-induced BET protein degradation as a therapy for castration-resistant prostate cancer

Proceedings of the National Academy of Sciences·2016·825 cit.·DOI

2

Recurrent somatic mutations in ACVR1 in pediatric midline high-grade astrocytoma

Nature Genetics·2014·464 cit.·DOI

3

Memory Impairment in Transgenic Alzheimer Mice Requires Cellular Prion Protein

Journal of Neuroscience·2010·406 cit.·DOI

4

EBV-driven LMP1 and IFN-γ up-regulate PD-L1 in nasopharyngeal carcinoma: Implications for oncotargeted therapy

Oncotarget·2014·386 cit.·DOI

5

Implications of the tumor immune microenvironment for staging and therapeutics

Modern Pathology·2017·364 cit.·DOI

6

Topological analysis reveals a PD-L1-associated microenvironmental niche for Reed-Sternberg cells in Hodgkin lymphoma

Blood·2017·332 cit.·DOI

7

Therapeutically activating RB: reestablishing cell cycle control in endocrine therapy-resistant breast cancer

Endocrine Related Cancer·2011·299 cit.·DOI

8

Cholesterol and ORP1L-mediated ER contact sites control autophagosome transport and fusion with the endocytic pathway

Nature Communications·2016·219 cit.·DOI

9

Long non-coding RNA PVT1 predicts poor prognosis and induces radioresistance by regulating DNA repair and cell apoptosis in nasopharyngeal carcinoma

Cell Death and Disease·2018·192 cit.·DOI

10

An <i>Acvr1</i> R206H knock-in mouse has fibrodysplasia ossificans progressiva

Journal of Bone and Mineral Research·2012·192 cit.·DOI

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