Cell Signaling Technology

LRP5 (D5G4) Rabbit Monoclonal Antibody

#5440 · $161

100
Citing papers
8.9k
Combined citations
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Research areas

Epigenetics and DNA Methylation11Autophagy in Disease and Therapy8Ubiquitin and proteasome pathways7Genetics and Neurodevelopmental Disorders5Alzheimer's disease research and treatments5Extracellular vesicles in disease5Cancer Immunotherapy and Biomarkers5Neuroinflammation and Neurodegeneration Mechanisms5

Papers citing this product

1
Exosome-shuttled miR-216a-5p from hypoxic preconditioned mesenchymal stem cells repair traumatic spinal cord injury by shifting microglial M1/M2 polarization
Journal of Neuroinflammation·2020·601 cit.·DOI
2
Critical roles of c-Jun signaling in regulation of NFAT family and RANKL-regulated osteoclast differentiation
Journal of Clinical Investigation·2004·417 cit.·DOI
3
Epigenetic Changes Induced by Reactive Oxygen Species in Hepatocellular Carcinoma: Methylation of the E-cadherin Promoter
Gastroenterology·2008·382 cit.·DOI
4
The Wnt5A/Protein Kinase C Pathway Mediates Motility in Melanoma Cells via the Inhibition of Metastasis Suppressors and Initiation of an Epithelial to Mesenchymal Transition
Journal of Biological Chemistry·2007·368 cit.·DOI
5
Proteotoxic Stress Induces Phosphorylation of p62/SQSTM1 by ULK1 to Regulate Selective Autophagic Clearance of Protein Aggregates
PLoS Genetics·2015·318 cit.·DOI
6
Plant and Animal Pathogen Recognition Receptors Signal through Non-RD Kinases
PLoS Pathogens·2006·277 cit.·DOI
7
Anti-inflammatory effects of BHBA in both in vivo and in vitro Parkinson’s disease models are mediated by GPR109A-dependent mechanisms
Journal of Neuroinflammation·2015·262 cit.·DOI
8
β-Hydroxybutyrate inhibits inflammasome activation to attenuate Alzheimer’s disease pathology
Journal of Neuroinflammation·2020·243 cit.·DOI
9
Regulation of SR protein phosphorylation and alternative splicing by modulating kinetic interactions of SRPK1 with molecular chaperones
Genes & Development·2009·238 cit.·DOI
10
Coronavirus infection and PARP expression dysregulate the NAD metabolome: An actionable component of innate immunity
Journal of Biological Chemistry·2020·196 cit.·DOI

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