Cell Signaling Technology

BRD4 (E2A7X) Rabbit Monoclonal Antibody (BSA and Azide Free)

#35632 · $865

9
Citing papers
327
Combined citations
Supplier page Find this product

Research areas

Renal Diseases and Glomerulopathies4Biomedical Research and Pathophysiology4Pancreatic function and diabetes3Chronic Kidney Disease and Diabetes2Diet, Metabolism, and Disease1Cardiac Fibrosis and Remodeling1Genetic and Kidney Cyst Diseases1Cancer, Hypoxia, and Metabolism1

Papers citing this product

1
A Role for ATP-Citrate Lyase, Malic Enzyme, and Pyruvate/Citrate Cycling in Glucose-induced Insulin Secretion
Journal of Biological Chemistry·2007·157 cit.·DOI
2
Pharmacological Targeting of the Mitochondrial Phosphatase PTPMT1
Journal of Pharmacology and Experimental Therapeutics·2010·70 cit.·DOI
3
Changes in Mitochondrial Carriers Exhibit Stress-Specific Signatures in INS-1Eβ-Cells Exposed to Glucose Versus Fatty Acids
PLoS ONE·2013·25 cit.·DOI
4
Genetic Ablation of Calcium-independent Phospholipase A2γ Induces Glomerular Injury in Mice
Journal of Biological Chemistry·2016·25 cit.·DOI
5
Mineralocorticoid receptor promotes cardiac macrophage inflammaging
Basic Research in Cardiology·2024·23 cit.·DOI
6
Genetic Ablation of Calcium-independent Phospholipase A2γ Exacerbates Glomerular Injury in Adriamycin Nephrosis in Mice
Scientific Reports·2019·16 cit.·DOI
7
Effects of hyperhomocysteinemia on follicular development and oocytes quality
iScience·2024·10 cit.·DOI
8
Genetic deletion of calcium-independent phospholipase A2γ protects mice from diabetic nephropathy
PLoS ONE·2024·1 cit.·DOI
9
Genetic Deletion of Calcium-independent Phospholipase A <sub>2</sub> γ Protects Mice from Diabetic Nephropathy
bioRxiv (Cold Spring Harbor Laboratory)·2024·DOI

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