Cell Signaling Technology

ELP3 (D5H12) Rabbit Monoclonal Antibody

#5728 · western blot · $161,351

100

Citing papers

31.4k

Combined citations

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Research areas

Autophagy in Disease and Therapy54 Neuroinflammation and Neurodegeneration Mechanisms15 Immune cells in cancer13 Endoplasmic Reticulum Stress and Disease12 Parkinson's Disease Mechanisms and Treatments9 Mitochondrial Function and Pathology8 Sirtuins and Resveratrol in Medicine8 Lysosomal Storage Disorders Research7

Papers citing this product

1

Inhibition of mTOR induces autophagy and reduces toxicity of polyglutamine expansions in fly and mouse models of Huntington disease

Nature Genetics·2004·2.3k cit.·DOI

2

An ATP-competitive Mammalian Target of Rapamycin Inhibitor Reveals Rapamycin-resistant Functions of mTORC1

Journal of Biological Chemistry·2009·1.8k cit.·DOI

3

Trehalose, a Novel mTOR-independent Autophagy Enhancer, Accelerates the Clearance of Mutant Huntingtin and α-Synuclein

Journal of Biological Chemistry·2006·1.1k cit.·DOI

4

ER stress (PERK/eIF2α phosphorylation) mediates the polyglutamine-induced LC3 conversion, an essential step for autophagy formation

Cell Death and Differentiation·2006·923 cit.·DOI

5

The unfolded protein response protects human tumor cells during hypoxia through regulation of the autophagy genes MAP1LC3B and ATG5

Journal of Clinical Investigation·2009·799 cit.·DOI

6

Toll‐like receptors control autophagy

The EMBO Journal·2008·727 cit.·DOI

7

Response to myocardial ischemia/reperfusion injury involves Bnip3 and autophagy

Cell Death and Differentiation·2006·605 cit.·DOI

8

Caloric restriction and resveratrol promote longevity through the Sirtuin-1-dependent induction of autophagy

Cell Death and Disease·2010·601 cit.·DOI

9

Acid sphingomyelinase activity triggers microparticle release from glial cells

The EMBO Journal·2009·586 cit.·DOI

10

CCL2 and Interleukin-6 Promote Survival of Human CD11b+ Peripheral Blood Mononuclear Cells and Induce M2-type Macrophage Polarization

Journal of Biological Chemistry·2009·571 cit.·DOI

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