Cell Signaling Technology

uPAR (D4Q5S) Rabbit Monoclonal Antibody

#12863 · western blot · $161,351

65
Evidence Score · Well-Established
Used by 141 independent labs across 182 papers. How we score this →
74
Citing papers
1.5k
Combined citations
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Research areas

Neuroscience and Neuropharmacology Research6Mitochondrial Function and Pathology6RNA modifications and cancer5Neurobiology and Insect Physiology Research5DNA Repair Mechanisms5Cancer-related molecular mechanisms research5Autophagy in Disease and Therapy4Ferroptosis and cancer prognosis3

Papers citing this product

1
Differential role of mitogen-activated protein kinase in three distinct phases of memory for sensitization in Aplysia.
PubMed·2003·117 cit.·DOI
2
Differential Role of Mitogen-Activated Protein Kinase in Three Distinct Phases of Memory for Sensitization in<i>Aplysia</i>
Journal of Neuroscience·2003·115 cit.·DOI
3
Intermediate-Term Memory for Site-Specific Sensitization in<i>Aplysia</i>Is Maintained by Persistent Activation of Protein Kinase C
Journal of Neuroscience·2004·90 cit.·DOI
4
Anandamide-induced cell death in primary neuronal cultures: role of calpain and caspase pathways
Cell Death and Differentiation·2004·81 cit.·DOI
5
Control of Programmed Cell Death by Distinct Electrical Activity Patterns
Cerebral Cortex·2010·66 cit.·DOI
6
Aspirin inhibits camptothecin-induced p21CIP1 levels and potentiates apoptosis in human breast cancer cells
International Journal of Oncology·2009·65 cit.·DOI
7
Concerted Regulation of cGMP and cAMP Phosphodiesterases in Early Cardiac Hypertrophy Induced by Angiotensin II
PLoS ONE·2010·60 cit.·DOI
8
The Circadian Clock Modulates Core Steps in Long-Term Memory Formation in<i>Aplysia</i>
Journal of Neuroscience·2006·54 cit.·DOI
9
Mechanism of Stimulation of Human DNA Ligase I by the Rad9-Rad1-Hus1 Checkpoint Complex
Journal of Biological Chemistry·2006·49 cit.·DOI
10
PI3K-resistant GSK3 controls adiponectin formation and protects from metabolic syndrome
Proceedings of the National Academy of Sciences·2016·48 cit.·DOI

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