Anandamide-induced cell death in primary neuronal cultures: role of calpain and caspase pathways
Vilen Movsesyan(Georgetown University), Alan I. Faden(University of Maryland, Baltimore)
Cited by 81
Related Papers
Activation of CPP32-Like Caspases Contributes to Neuronal Apoptosis and Neurological Dysfunction after Traumatic Brain Injury
|Journal of Neuroscience|1997|583
Differential Expression of Apoptotic Protease-Activating Factor-1 and Caspase-3 Genes and Susceptibility to Apoptosis during Brain Development and after Traumatic Brain Injury
|Journal of Neuroscience|2001|263
Downregulation of miR-23a and miR-27a following Experimental Traumatic Brain Injury Induces Neuronal Cell Death through Activation of Proapoptotic Bcl-2 Proteins
|Journal of Neuroscience|2014|156
BOK and NOXA Are Essential Mediators of p53-dependent Apoptosis
|Journal of Biological Chemistry|2004|153
The Role of CED-3-Related Cysteine Proteases in Apoptosis of Cerebellar Granule Cells
|Journal of Neuroscience|1997|130