Cell Signaling Technology

DRP1 (D6C7) Rabbit Monoclonal Antibody

#8570 · $174

100
Citing papers
8.5k
Combined citations
Supplier page Find this product

Research areas

Mitochondrial Function and Pathology26Autophagy in Disease and Therapy16Epigenetics and DNA Methylation12Cancer-related molecular mechanisms research8FOXO transcription factor regulation8ATP Synthase and ATPases Research6Adipose Tissue and Metabolism6RNA modifications and cancer6

Papers citing this product

1
Mitophagy inhibits amyloid-β and tau pathology and reverses cognitive deficits in models of Alzheimer’s disease
Nature Neuroscience·2019·1.7k cit.·DOI
2
Somatic mutations at EZH2 Y641 act dominantly through a mechanism of selectively altered PRC2 catalytic activity, to increase H3K27 trimethylation
Blood·2010·597 cit.·DOI
3
Long non-coding RNA XIST regulates gastric cancer progression by acting as a molecular sponge of miR-101 to modulate EZH2 expression
Journal of Experimental & Clinical Cancer Research·2016·264 cit.·DOI
4
Urea-induced ROS generation causes insulin resistance in mice with chronic renal failure
Journal of Clinical Investigation·2009·229 cit.·DOI
5
The BDNF effects on dendritic spines of mature hippocampal neurons depend on neuronal activity
Frontiers in Synaptic Neuroscience·2014·170 cit.·DOI
6
Synaptic Activity Controls Dendritic Spine Morphology by Modulating eEF2-Dependent BDNF Synthesis
Journal of Neuroscience·2010·158 cit.·DOI
7
Genome-Wide Expression Analysis of Middle Eastern Colorectal Cancer Reveals FOXM1 as a Novel Target for Cancer Therapy
American Journal Of Pathology·2011·155 cit.·DOI
8
A cell-penetrating ARF peptide inhibitor of FoxM1 in mouse hepatocellular carcinoma treatment
Journal of Clinical Investigation·2006·155 cit.·DOI
9
ROS-mediated activation and mitochondrial translocation of CaMKII contributes to Drp1-dependent mitochondrial fission and apoptosis in triple-negative breast cancer cells by isorhamnetin and chloroquine
Journal of Experimental & Clinical Cancer Research·2019·139 cit.·DOI
10
Insulin and IGF-1 improve mitochondrial function in a PI-3K/Akt-dependent manner and reduce mitochondrial generation of reactive oxygen species in Huntington’s disease knock-in striatal cells
Free Radical Biology and Medicine·2014·138 cit.·DOI

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DRP1 (D6C7) Rabbit Monoclonal Antibody — Evidence — PubIndex