Mitophagy inhibits amyloid-β and tau pathology and reverses cognitive deficits in models of Alzheimer’s disease
Evandro Fei Fang(National Institutes of Health), Vilhelm A. Bohr(University of Copenhagen), Md Mahdi Hasan‐Olive(University of Oslo), Sofie Lautrup(University of Oslo), Domenica Caponio(University of Oslo), M. Zameel Cader(Oxford BioMedica (United Kingdom)), Xiuli Dan(University of Minnesota), Nigel H. Greig(National Institutes of Health), Tormod Fladby(University of Oslo), Bryan A. Adriaanse(University of Oxford), Mansour Akbari(University of Copenhagen), Nektarios Tavernarakis(University of Crete), Paula Rocktäschel(John Radcliffe Hospital), Jesse S. Kerr(National Institutes of Health), Hilde Nilsen(Oslo University Hospital), Mark P. Mattson(Johns Hopkins University), Beimeng Yang(National Institute on Aging), Konstantinos Palikaras(National and Kapodistrian University of Athens), Yujun Hou(Chengdu University of Traditional Chinese Medicine), Deborah L. Croteau(National Institutes of Health)
Cited by 1,687
Related Papers
Genome-wide meta-analysis identifies new loci and functional pathways influencing Alzheimer’s disease risk
|Nature Genetics|2019|2.5k
Molecular definitions of autophagy and related processes
|The EMBO Journal|2017|1.6k
Brain energy rescue: an emerging therapeutic concept for neurodegenerative disorders of ageing
|Nature Reviews Drug Discovery|2020|982
Mitophagy and Alzheimer’s Disease: Cellular and Molecular Mechanisms
|Trends in Neurosciences|2017|861