Cell Signaling Technology

PDHK1 (C47H1) Rabbit Monoclonal Antibody

#3820 · $152,351

100
Citing papers
12.7k
Combined citations
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Research areas

Cancer, Hypoxia, and Metabolism12Metabolism, Diabetes, and Cancer11Adipose Tissue and Metabolism9Immune Cell Function and Interaction7Immune Response and Inflammation6Autophagy in Disease and Therapy6Inflammasome and immune disorders5Adenosine and Purinergic Signaling5

Papers citing this product

1
Glucose Uptake Is Limiting in T Cell Activation and Requires CD28-Mediated Akt-Dependent and Independent Pathways
The Journal of Immunology·2008·836 cit.·DOI
2
BAF complex vulnerabilities in cancer demonstrated via structure-based PROTAC design
Nature Chemical Biology·2019·571 cit.·DOI
3
Production and Release of Neuroprotective Tumor Necrosis Factor by P2X<sub>7</sub>Receptor-Activated Microglia
Journal of Neuroscience·2004·391 cit.·DOI
4
New approaches to molecular cancer therapeutics
Nature Chemical Biology·2006·387 cit.·DOI
5
O-GlcNAcylation of PGK1 coordinates glycolysis and TCA cycle to promote tumor growth
Nature Communications·2020·345 cit.·DOI
6
Acylated and Unacylated Ghrelin Promote Proliferation and Inhibit Apoptosis of Pancreatic β-Cells and Human Islets: Involvement of 3′,5′-Cyclic Adenosine Monophosphate/Protein Kinase A, Extracellular Signal-Regulated Kinase 1/2, and Phosphatidyl Inositol 3-Kinase/Akt Signaling
Endocrinology·2006·290 cit.·DOI
7
Downregulation of GSDMD attenuates tumor proliferation via the intrinsic mitochondrial apoptotic pathway and inhibition of EGFR/Akt signaling and predicts a good prognosis in non‑small cell lung cancer
Oncology Reports·2018·279 cit.·DOI
8
Tumor-Infiltrating Programmed Death Receptor-1+ Dendritic Cells Mediate Immune Suppression in Ovarian Cancer
The Journal of Immunology·2011·241 cit.·DOI
9
ROS-dependent S-palmitoylation activates cleaved and intact gasdermin D
Nature·2024·217 cit.·DOI
10
The docking protein Gab1 is the primary mediator of EGF-stimulated activation of the PI-3K/Akt cell survival pathway
BMC Biology·2004·210 cit.·DOI

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