Cell Signaling Technology

Presenilin 2 (D30G3) Rabbit Monoclonal Antibody

#9979 · $152

100
Citing papers
5.1k
Combined citations
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Research areas

Alzheimer's disease research and treatments15Autophagy in Disease and Therapy11Ubiquitin and proteasome pathways9Cancer Cells and Metastasis7Cancer-related molecular mechanisms research6Endoplasmic Reticulum Stress and Disease6Circular RNAs in diseases5Neuroinflammation and Neurodegeneration Mechanisms5

Papers citing this product

1
Metabolic reprogramming during neuronal differentiation
Cell Death and Differentiation·2016·265 cit.·DOI
2
Salidroside Ameliorates Renal Interstitial Fibrosis by Inhibiting the TLR4/NF-κB and MAPK Signaling Pathways
International Journal of Molecular Sciences·2019·201 cit.·DOI
3
A novel rapamycin analog is highly selective for mTORC1 in vivo
Nature Communications·2019·196 cit.·DOI
4
Genetic Reduction of Mammalian Target of Rapamycin Ameliorates Alzheimer's Disease-Like Cognitive and Pathological Deficits by Restoring Hippocampal Gene Expression Signature
Journal of Neuroscience·2014·187 cit.·DOI
5
A Becn1 mutation mediates hyperactive autophagic sequestration of amyloid oligomers and improved cognition in Alzheimer's disease
PLoS Genetics·2017·160 cit.·DOI
6
Low temperature inhibits anthocyanin accumulation in strawberry fruit by activating FvMAPK3-induced phosphorylation of FvMYB10 and degradation of Chalcone Synthase 1
The Plant Cell·2022·146 cit.·DOI
7
An inhibitor of the proteasomal deubiquitinating enzyme USP14 induces tau elimination in cultured neurons
Journal of Biological Chemistry·2017·144 cit.·DOI
8
Inhibition of mTOR protects the blood-brain barrier in models of Alzheimer’s disease and vascular cognitive impairment
American Journal of Physiology-Heart and Circulatory Physiology·2017·134 cit.·DOI
9
The mTOR Inhibitor Rapamycin Mitigates Perforant Pathway Neurodegeneration and Synapse Loss in a Mouse Model of Early-Stage Alzheimer-Type Tauopathy
PLoS ONE·2015·123 cit.·DOI
10
A novel pathway down-modulating T cell activation involves HPK-1–dependent recruitment of 14-3-3 proteins on SLP-76
The Journal of Experimental Medicine·2007·107 cit.·DOI

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