Abcam

Anti-Caspase-3 antibody

#ab13847 · western blot

200
Citing papers
14.2k
Combined citations
Supplier page Find this product

Research areas

MicroRNA in disease regulation13Cancer-related molecular mechanisms research10Circular RNAs in diseases8Extracellular vesicles in disease6Mitochondrial Function and Pathology5Autophagy in Disease and Therapy4Immune cells in cancer4Endoplasmic Reticulum Stress and Disease3

Papers citing this product

1
Endothelial to mesenchymal transition is common in atherosclerotic lesions and is associated with plaque instability
Nature Communications·2016·596 cit.·DOI
2
microRNA-21-5p dysregulation in exosomes derived from heart failure patients impairs regenerative potential
Journal of Clinical Investigation·2019·310 cit.·DOI
3
Sustained production of ROS triggers compensatory proliferation and is required for regeneration to proceed
Scientific Reports·2013·304 cit.·DOI
4
Reversing drug resistance of soft tumor-repopulating cells by tumor cell-derived chemotherapeutic microparticles
Cell Research·2016·282 cit.·DOI
5
CircRNA-ENO1 promoted glycolysis and tumor progression in lung adenocarcinoma through upregulating its host gene ENO1
Cell Death and Disease·2019·280 cit.·DOI
6
Inhibiting macrophage proliferation suppresses atherosclerotic plaque inflammation
Science Advances·2015·227 cit.·DOI
7
Prevotellaceae produces butyrate to alleviate PD-1/PD-L1 inhibitor-related cardiotoxicity via PPARα-CYP4X1 axis in colonic macrophages
Journal of Experimental & Clinical Cancer Research·2022·211 cit.·DOI
8
Exosomes derived from cancer stem cells of gemcitabine-resistant pancreatic cancer cells enhance drug resistance by delivering miR-210
Cellular Oncology·2019·195 cit.·DOI
9
Excess heme upregulates heme oxygenase 1 and promotes cardiac ferroptosis in mice with sickle cell disease
Blood·2021·177 cit.·DOI
10
MiR‐29b‐3p promotes chondrocyte apoptosis and facilitates the occurrence and development of osteoarthritis by targeting <scp>PGRN</scp>
Journal of Cellular and Molecular Medicine·2017·173 cit.·DOI

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