Cell Signaling Technology

Phospho-SATB1 (Ser47) Antibody

#4028 · $362

100
Citing papers
10.5k
Combined citations
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Research areas

Metabolism, Diabetes, and Cancer9Cancer-related gene regulation7TGF-β signaling in diseases7Pancreatic function and diabetes7Ubiquitin and proteasome pathways6Epigenetics and DNA Methylation6Angiogenesis and VEGF in Cancer5Cancer, Hypoxia, and Metabolism5

Papers citing this product

1
Proteogenomic Characterization Reveals Therapeutic Vulnerabilities in Lung Adenocarcinoma
Cell·2020·811 cit.·DOI
2
Aβ Oligomers Cause Localized Ca <sup>2+</sup> Elevation, Missorting of Endogenous Tau into Dendrites, Tau Phosphorylation, and Destruction of Microtubules and Spines
Journal of Neuroscience·2010·677 cit.·DOI
3
Increase in ALK1/ALK5 Ratio as a Cause for Elevated MMP-13 Expression in Osteoarthritis in Humans and Mice
The Journal of Immunology·2009·315 cit.·DOI
4
Missorting of Tau in Neurons Causes Degeneration of Synapses That Can Be Rescued by the Kinase MARK2/Par-1
Journal of Neuroscience·2007·287 cit.·DOI
5
Activation of  -Glucocerebrosidase Reduces Pathological  -Synuclein and Restores Lysosomal Function in Parkinson's Patient Midbrain Neurons
Journal of Neuroscience·2016·275 cit.·DOI
6
TGFβ1 secreted by cancer-associated fibroblasts induces epithelial-mesenchymal transition of bladder cancer cells through lncRNA-ZEB2NAT
Scientific Reports·2015·251 cit.·DOI
7
A New Glucocerebrosidase Chaperone Reduces  -Synuclein and Glycolipid Levels in iPSC-Derived Dopaminergic Neurons from Patients with Gaucher Disease and Parkinsonism
Journal of Neuroscience·2016·229 cit.·DOI
8
ChIP-seq reveals cell type-specific binding patterns of BMP-specific Smads and a novel binding motif
Nucleic Acids Research·2011·226 cit.·DOI
9
Pseudomonas aeruginosa Pili and Flagella Mediate Distinct Binding and Signaling Events at the Apical and Basolateral Surface of Airway Epithelium
PLoS Pathogens·2012·209 cit.·DOI
10
Phosphoinositide-3 Kinase-Akt Pathway Controls Cellular Entry of Ebola Virus
PLoS Pathogens·2008·197 cit.·DOI