Minute amounts of helicase-deficient truncated RECQL4 are sufficient for DNA replication
Paula Armina V. Buco(Hudson Institute), Carl R. Walkley(The University of Melbourne)
Cited by 2
Related Papers
BET inhibitors induce apoptosis through a MYC independent mechanism and synergise with CDK inhibitors to kill osteosarcoma cells
|Scientific Reports|2015|124
The phenotype of the most common human ADAR1p150 Zα mutation P193A in mice is partially penetrant
|EMBO Reports|2023|29
Tolerance to sustained activation of the cAMP/Creb pathway activity in osteoblastic cells is enabled by loss of p53
|Cell Death and Disease|2018|15
Cell death following the loss of ADAR1 mediated A-to-I RNA editing is not effected by the intrinsic apoptosis pathway
|Cell Death and Disease|2019|15
The most common human ADAR1p150 Zα domain mutation P193A is well tolerated in mice and does not activate the integrated stress response pathway
|bioRxiv (Cold Spring Harbor Laboratory)|2022|2