Tolerance to sustained activation of the cAMP/Creb pathway activity in osteoblastic cells is enabled by loss of p53
Mannu Walia(St Vincents Institute of Medical Research), Carl R. Walkley(The University of Melbourne)
Cited by 15
Related Papers
BET inhibitors induce apoptosis through a MYC independent mechanism and synergise with CDK inhibitors to kill osteosarcoma cells
|Scientific Reports|2015|124
The phenotype of the most common human ADAR1p150 Zα mutation P193A in mice is partially penetrant
|EMBO Reports|2023|29
Cell death following the loss of ADAR1 mediated A-to-I RNA editing is not effected by the intrinsic apoptosis pathway
|Cell Death and Disease|2019|15
The most common human ADAR1p150 Zα domain mutation P193A is well tolerated in mice and does not activate the integrated stress response pathway
|bioRxiv (Cold Spring Harbor Laboratory)|2022|2
Minute amounts of helicase-deficient truncated RECQL4 are sufficient for DNA replication
|bioRxiv (Cold Spring Harbor Laboratory)|2025|2