Ca+2 and Nε-lysine acetylation regulate the CALR-ATG9A interaction in the lumen of the endoplasmic reticulum
Megan M. Braun(University of Wisconsin–Madison), Luigi Puglielli(University of Wisconsin System)
Cited by 0
Related Papers
Deficient Import of Acetyl-CoA into the ER Lumen Causes Neurodegeneration and Propensity to Infections, Inflammation, and Cancer
|Journal of Neuroscience|2014|60
Increased expression of AT-1/SLC33A1 causes an autistic-like phenotype in mice by affecting dendritic branching and spine formation
|The Journal of Experimental Medicine|2016|39
Increased expression of SLC25A1/CIC causes an autistic-like phenotype with altered neuron morphology
|Brain|2021|24