Deficient Import of Acetyl-CoA into the ER Lumen Causes Neurodegeneration and Propensity to Infections, Inflammation, and Cancer
Yajing Peng, Luigi Puglielli(University of Wisconsin System)
Cited by 60
Related Papers
Increased expression of AT-1/SLC33A1 causes an autistic-like phenotype in mice by affecting dendritic branching and spine formation
|The Journal of Experimental Medicine|2016|39
Increased expression of SLC25A1/CIC causes an autistic-like phenotype with altered neuron morphology
|Brain|2021|24
Endoplasmic reticulum acetyltransferases Atase1 and Atase2 differentially regulate reticulophagy, macroautophagy and cellular acetyl-CoA metabolism
|Communications Biology|2021|22
SLC13A5/sodium-citrate co-transporter overexpression causes disrupted white matter integrity and an autistic-like phenotype
|Brain Communications|2022|22
The citrate transporters SLC13A5 and SLC25A1 elicit different metabolic responses and phenotypes in the mouse
|Communications Biology|2023|19