Shaping cardiac destiny: the role of post-translational modifications on endoplasmic reticulum – mitochondria crosstalk in cardiac remodeling
Xiaohan Zhang(Chinese Academy of Medical Sciences & Peking Union Medical College), Bai Du(Chinese Academy of Medical Sciences & Peking Union Medical College), Yuanhui Hu(Chinese Academy of Medical Sciences & Peking Union Medical College), Zezhen Guo(Macquarie University), Yihang Du(Chinese Academy of Medical Sciences & Peking Union Medical College), Shuqing Shi(Chinese Academy of Medical Sciences & Peking Union Medical College), Xing Chang(Chinese Academy of Medical Sciences & Peking Union Medical College), Ruoning Chai(China Academy of Chinese Medical Sciences), Huan Wang(Science and Technology Department of Sichuan Province), Chenglin Duan(Chinese Academy of Medical Sciences & Peking Union Medical College)
Cited by 7
Related Papers
Emerging insights into the pathogenesis and therapeutic strategies for vascular endothelial injury-associated diseases: focus on mitochondrial dysfunction
|Angiogenesis|2024|215
Nuclear receptor subfamily 4 group A member 1 promotes myocardial ischemia/reperfusion injury through inducing mitochondrial fission factor-mediated mitochondrial fragmentation and inhibiting FUN14 domain containing 1-depedent mitophagy
|International Journal of Biological Sciences|2024|165
New insights into the role of mitochondrial metabolic dysregulation and immune infiltration in septic cardiomyopathy by integrated bioinformatics analysis and experimental validation
|Cellular & Molecular Biology Letters|2024|148
Advances in pathogenesis and treatment of vascular endothelial injury-related diseases mediated by mitochondrial abnormality
|Frontiers in Pharmacology|2024|125
Targeting mitochondria by lipid-selenium conjugate drug results in malate/fumarate exhaustion and induces mitophagy-mediated necroptosis suppression
|International Journal of Biological Sciences|2024|115