New insights into the role of mitochondrial metabolic dysregulation and immune infiltration in septic cardiomyopathy by integrated bioinformatics analysis and experimental validation

Yukun Li(Capital Medical University), Xing Chang(Chinese Academy of Medical Sciences & Peking Union Medical College), Hao Zhou(Chinese PLA General Hospital), Ruibin Li(Soochow University), Jiachi Yu
Cellular & Molecular Biology Letters
January 30, 2024
10.1186/s11658-024-00536-2
Cited by 148

Related Papers

DUSP1 alleviates cardiac ischemia/reperfusion injury by suppressing the Mff-required mitochondrial fission and Bnip3-related mitophagy via the JNK pathways
|Redox Biology|2017|459
Antibacterial Nanomaterials: Mechanisms, Impacts on Antimicrobial Resistance and Design Principles
|Angewandte Chemie International Edition|2023|439
Melatonin protects cardiac microvasculature against ischemia/reperfusion injury via suppression of mitochondrial fission‐<scp>VDAC</scp>1‐<scp>HK</scp>2‐<scp>mPTP</scp>‐mitophagy axis
|Journal of Pineal Research|2017|374
Emerging insights into the pathogenesis and therapeutic strategies for vascular endothelial injury-associated diseases: focus on mitochondrial dysfunction
|Angiogenesis|2024|215
Nuclear receptor subfamily 4 group A member 1 promotes myocardial ischemia/reperfusion injury through inducing mitochondrial fission factor-mediated mitochondrial fragmentation and inhibiting FUN14 domain containing 1-depedent mitophagy
|International Journal of Biological Sciences|2024|165