Evolving antibody response to SARS-CoV-2 antigenic shift from XBB to JN.1

Fanchong Jian(Pioneer (United States)), Jing Wang(Pioneer (United States)), Ayijiang Yisimayi(Pioneer (United States)), Weiliang Song(Pioneer (United States)), Yanli Xu(Capital Medical University), Xiaosu Chen(Nankai University), Xiao Niu(Peking University), Sijie Yang(Peking University), Yuanling Yu(Beijing Tsinghua Chang Gung Hospital), Peng Wang(Beijing Tsinghua Chang Gung Hospital), Haiyan Sun(Beijing Tsinghua Chang Gung Hospital), Lingling Yu(Beijing Tsinghua Chang Gung Hospital), Jing Wang(Peking University), Yao Wang(National Institutes for Food and Drug Control), Ran An(Peking University), Wenjing Wang(Beijing Tsinghua Chang Gung Hospital), Miaomiao Ma(Pioneer (United States)), Tianhe Xiao(Peking University), Qingqing Gu(Beijing Tsinghua Chang Gung Hospital), Fei Shao(Beijing Tsinghua Chang Gung Hospital), Youchun Wang(National Institutes for Food and Drug Control), Zhongyang Shen(Nankai University), Ronghua Jin(Capital Medical University), Yunlong Cao(Peking University)
bioRxiv (Cold Spring Harbor Laboratory)
April 22, 2024
Cited by 29Open Access
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Abstract

Abstract The continuous evolution of SARS-CoV-2, particularly the emergence of the BA.2.86/JN.1 lineage replacing XBB lineages, necessitates re-evaluation of current vaccine compositions. Here, we provide a comprehensive analysis of the humoral immune response to XBB and JN.1 human exposures, emphasizing the need for JN.1-lineage-based boosters. We demonstrate the antigenic distinctiveness of XBB and JN.1 lineages in SARS-CoV-2-naive individuals but not in those with prior vaccinations or infections, and JN.1 infection elicits superior plasma neutralization titers against its subvariants. We highlight the strong immune evasion and receptor binding capability of KP.3, supporting its foreseeable prevalence. Extensive analysis of the BCR repertoire, isolating ∼2000 RBD-specific monoclonal antibodies (mAbs) with their targeting epitopes characterized by deep mutational scanning (DMS), underscores the systematic superiority of JN.1-elicited memory B cells (MBCs). Notably, Class 1 IGHV3-53/3-66-derived neutralizing antibodies (NAbs) contribute majorly within wildtype (WT)-reactive NAbs against JN.1. However, KP.2 and KP.3 evade a substantial subset of them, even those induced by JN.1, advocating for booster updates to KP.3 for optimized enrichment. JN.1-induced Omicron-specific antibodies also demonstrate high potency across all Omicron lineages. Escape hotspots of these NAbs have mainly been mutated in Omicron RBD, resulting in higher immune barrier to escape, considering the probable recovery of previously escaped NAbs. Additionally, the prevalence of broadly reactive IGHV3-53/3-66- encoding antibodies and MBCs, and their capability of competing with all Omicron-specific NAbs suggests their inhibitory role on the de novo activation of Omicron-specific naive B cells, potentially explaining the heavy immune imprinting in mRNA-vaccinated individuals. These findings delineate the evolving antibody response to Omicron antigenic shift from XBB to JN.1, and highlight the importance of developing JN.1 lineage, especially KP.3-based vaccine boosters, to enhance humoral immunity against current and future SARS-CoV-2 variants.


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