TMIC-04. IMMUNE PROFILING OF PEDIATRIC ONCOHISTONE GLIOMAS REVEALS DIVERSE MYELOID POPULATIONS AND TUMOR-PROMOTING BEHAVIORS
Augusto Faria Andrade(McGill University Health Centre), Nada Jabado(McGill University), Daniela F. Quail(McGill University Health Centre), Geoffroy Danieau(Inserm), Caterina Russo(McGill University Health Centre), Emily M. Nakada(University of Vermont), Valérie Larouche(Université Laval), Claudia L. Kleinman(Jewish General Hospital), Arne Gehlhaar(Heidelberg University), Danai G. Topouza(McGill University), Damien Faury(Montreal Children's Hospital), Benjamin Ellezam(Centre Hospitalier Universitaire Sainte-Justine), Eduardo Gonzalez Santiago(Yale University), Liza Konnikova(Yale University), Sameer Agnihotri(University of Pittsburgh), Nikoleta Juretic(McGill Genome Centre), Michael McNicholas(Brain Tumour Research), Antonella De Cola(Brain Tumour Research), Selin Jessa(Stanford University), Brian Krug(McGill University), Manav Pathania(Brain Tumour Research), Roy Dudley(McGill University Health Centre), Alexander G. Weil(McGill University), Logan A. Walsh(Institute for Research in Immunology and Cancer), Bhavyaa Chandarana(McGill University), Qing Wu(McGill University Health Centre), Jason Karamchandani(Montreal Neurological Institute and Hospital), Yuhong Wei(McGill University), Michele Zeinieh(McGill University Health Centre)
Cited by 0
Related Papers
Intertumoral Heterogeneity within Medulloblastoma Subgroups
|Cancer Cell|2017|1.3k
K27M mutation in histone H3.3 defines clinically and biologically distinct subgroups of pediatric diffuse intrinsic pontine gliomas
|Acta Neuropathologica|2012|1k
Individual intestinal symbionts induce a distinct population of RORγ <sup>+</sup> regulatory T cells
|Science|2015|886
Immune Checkpoint Inhibition for Hypermutant Glioblastoma Multiforme Resulting From Germline Biallelic Mismatch Repair Deficiency
|Journal of Clinical Oncology|2016|867