Endogenous retroviruses and TDP-43 proteinopathy form a sustaining feedback driving intercellular spread of Drosophila neurodegeneration
Yung‐Heng Chang(Stony Brook School), Josh Dubnau(Cold Spring Harbor Laboratory)
Cited by 44
Related Papers
Postmortem Cortex Samples Identify Distinct Molecular Subtypes of ALS: Retrotransposon Activation, Oxidative Stress, and Activated Glia
|Cell Reports|2019|333
Unexpected similarities between C9ORF72 and sporadic forms of ALS/FTD suggest a common disease mechanism
|eLife|2018|87
Nuclear Transcriptomes of the Seven Neuronal Cell Types That Constitute the <i>Drosophila</i> Mushroom Bodies
|G3 Genes Genomes Genetics|2018|64
Cell environment shapes TDP-43 function with implications in neuronal and muscle disease
|Communications Biology|2022|52