Unexpected similarities between C9ORF72 and sporadic forms of ALS/FTD suggest a common disease mechanism

Erin G. Conlon(Columbia University), James L. Manley(Columbia University), James A. Gregory(New York Genome Center), Efthimios Dardiotis(University of Thessaly), Dhruv Sareen(Cedars-Sinai Medical Center), Julia Davis‐Porada(Columbia University), Kristy Kang(New York Genome Center), Molly Hammell(Georgetown University), Frank Baas(The Netherlands Cancer Institute), Zachary Simmons(Pennsylvania State University), James D. Berry(Harvard University), Delphine Fagegaltier(Cold Spring Harbor Laboratory), James R. Broach(Princeton University), Eran Hornstein(Weizmann Institute of Science), Leslie M. Thompson(University of California, Irvine), Ximena Arcila-Londono(Henry Ford Health System), Ernest Fraenkel(Broad Institute), Hemali Phatnani(Duke Medical Center), Jonathan Mill(King's College London), Pietro Fratta, Noah Zaitlen(University of California, San Francisco), Edward B. Lee(University of Pennsylvania), Siddharthan Chandran(University of Cambridge), Phaedra Agius(New York Genome Center), Gregory A. Cox(Jackson Laboratory), Andrea Malaspina(Queen Mary University of London), Steven Finkbeiner(University of California, San Francisco), Nikolaos A. Patsopoulos(Broad Institute), Josh Dubnau(Stony Brook School), Duyang Kim(New York Genome Center), Isabel Hubbard(New York Genome Center), Neil A. Shneider(Columbia University Irving Medical Center), Lyle W. Ostrow(Johns Hopkins University), Daniel J. MacGowan(Icahn School of Medicine at Mount Sinai), Suvankar Pal(Mott MacDonald (United Kingdom)), Justin Kwan(National Institute of Neurological Disorders and Stroke), Avindra Nath(National Institutes of Health), Vivianna M. Van Deerlin(University of Pennsylvania), Terry Heiman‐Patterson(Temple University)
eLife
July 13, 2018
Cited by 87


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