Urate Transporter ABCG2 Function and Asymptomatic Hyperuricemia: A Retrospective Cohort Study of CKD Progression
Yuki Ohashi(Tokyo University of Pharmacy and Life Sciences), Kimiyoshi Ichida(Tokyo University of Pharmacy and Life Sciences), Yusuke Kawamura(National Defense Medical College), Mai Sekine(Nippon Veterinary and Life Science University), Takashi Yokoo(Jikei University School of Medicine), Akiyoshi Nakayama(National Defense Medical College), Hirotaka Matsuo(National Defense Medical College), Satoru Kuriyama(Jikei University School of Medicine), Takahiro Nakamura(Kyoto University), Yu Toyoda(University of Tokyo Hospital), Tomoko Nakano(National Tax College), Tappei Takada(University of Tokyo Hospital)
Cited by 33
Related Papers
Decreased extra-renal urate excretion is a common cause of hyperuricemia
|Nature Communications|2012|703
Hepatocyte Growth Factor Induces Gefitinib Resistance of Lung Adenocarcinoma with Epidermal Growth Factor Receptor–Activating Mutations
|Cancer Research|2008|642
Injection of Cultured Cells with a ROCK Inhibitor for Bullous Keratopathy
|New England Journal of Medicine|2018|551
Common Defects of ABCG2, a High-Capacity Urate Exporter, Cause Gout: A Function-Based Genetic Analysis in a Japanese Population
|Science Translational Medicine|2009|438
Mutations in Glucose Transporter 9 Gene SLC2A9 Cause Renal Hypouricemia
|The American Journal of Human Genetics|2008|386