SMARCA4 inactivation promotes lineage-specific transformation and early metastatic features in the lung
Carla P. Concepcion(Massachusetts Institute of Technology), Tyler Jacks(Howard Hughes Medical Institute), Adam J. Schoenfeld(Memorial Sloan Kettering Cancer Center), Francisco J. Sánchez‐Rivera(Memorial Sloan Kettering Cancer Center), Lindsay M. LaFave(Harvard Stem Cell Institute), Charles M. Rudin(Memorial Sloan Kettering Cancer Center), Roderick T. Bronson(Harvard University), Carla F. Kim(Boston Children's Hospital), Lydia P. DeAngelo(Massachusetts Institute of Technology), Vinay K. Kartha(Harvard Stem Cell Institute), Jason D. Buenrostro(Broad Institute), Gregory J. Riely(Memorial Sloan Kettering Cancer Center), Manav Gupta(Boston Children's Hospital), Isabella Del Priore(Massachusetts Institute of Technology), Jonathan Y. Kim(Massachusetts Institute of Technology), Manyuan Liu(Massachusetts Institute of Technology), Natasha Rekhtman(Albert Einstein College of Medicine), Manon Miller(Massachusetts Institute of Technology), Kevin Meli(Massachusetts Institute of Technology), Sai Ma(Broad Institute), Aviv Regev(Broad Institute), Arjun Bhutkar(Massachusetts Institute of Technology), Peter M.K. Westcott(Cold Spring Harbor Laboratory)
DSpace@MIT (Massachusetts Institute of Technology)
January 1, 2022
Cited by 86
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