SMARCA4 inactivation promotes lineage-specific transformation and early metastatic features in the lung
Carla P. Concepcion(Columbia University), Tyler Jacks(Massachusetts Institute of Technology), Adam J. Schoenfeld(Memorial Sloan Kettering Cancer Center), Francisco J. Sánchez‐Rivera(Howard Hughes Medical Institute), Lindsay M. LaFave(Harvard Stem Cell Institute), Charles M. Rudin(Memorial Sloan Kettering Cancer Center), Roderick T. Bronson(Harvard University), Carla F. Kim(Harvard Stem Cell Institute), Lydia P. DeAngelo(Massachusetts Institute of Technology), Vinay K. Kartha(Harvard Stem Cell Institute), Jason D. Buenrostro(Broad Institute), Gregory J. Riely(Memorial Sloan Kettering Cancer Center), Manav Gupta(Boston Children's Hospital), Isabella Del Priore(Beth Israel Deaconess Medical Center), Jonathan Y. Kim(Massachusetts Institute of Technology), Manyuan Liu(Massachusetts Institute of Technology), Natasha Rekhtman(Memorial Sloan Kettering Cancer Center), Manon Miller(Massachusetts Institute of Technology), Kevin Meli(Massachusetts Institute of Technology), Sai Ma(Broad Institute), Aviv Regev(Broad Institute), Arjun Bhutkar(Massachusetts Institute of Technology), Peter M.K. Westcott(Cold Spring Harbor Laboratory)
DSpace@MIT (Massachusetts Institute of Technology)
January 1, 2022
Cited by 86
Related Papers
Full-length transcriptome assembly from RNA-Seq data without a reference genome
|Nature Biotechnology|2011|22.7k
De novo transcript sequence reconstruction from RNA-seq using the Trinity platform for reference generation and analysis
|Nature Protocols|2013|9.1k
Spatial reconstruction of single-cell gene expression data
|Nature Biotechnology|2015|7.6k