Ligands binding to the prion protein induce its proteolytic release with therapeutic potential in neurodegenerative proteinopathies
Luise Linsenmeier(Universität Hamburg), Markus Glatzel(Universität Hamburg), Michaela Schweizer(Universität Hamburg), Ladan Amin(Boston University), Hermann C. Altmeppen(Universität Hamburg), Feizhi Song(Universität Hamburg), Sebastian Jung(Ruhr University Bochum), Adriano Aguzzi(University Hospital of Zurich), Simrika Thapa(University of Calgary), Paul Säftig(Christian-Albrechts-Universität zu Kiel), David A. Harris(Boston University), Hermann Schätzl(University of Calgary), Behnam Mohammadi(Universität Hamburg), Julia Bär(Universität Hamburg), Mohsin Shafiq(Universität Hamburg), Tania Massignan(University of Trento), Emiliano Biasini(University of Trento), Simone Hornemann(University of Zurich), Sabine Gilch(University of Calgary), Stefano Da Vela(European Molecular Biology Laboratory), Amulya Nidhi Shrivastava(Institut de Biologie de l'École Normale Supérieure), Marina Mikhaylova(Universität Hamburg), Berta Puig(Universität Hamburg), Antoine Triller(Centre National de la Recherche Scientifique), Karl Frontzek(University Hospital of Zurich), Federica Mazzola(Università della Svizzera italiana), Dmitri I. Svergun(European Molecular Biology Laboratory), Markus Daμμe(Christian-Albrechts-Universität zu Kiel), Alexander Schwarz(Universität Hamburg), Luca Varani(Institute for Research in Biomedicine), Jörg Tatzelt(Ruhr University Bochum), Matthias Schmitz(University of Göttingen), Angela da Silva Correia(German Center for Neurodegenerative Diseases), Inga Zerr(Universitätsmedizin Göttingen)
Cited by 33
Related Papers
The TREM2-APOE Pathway Drives the Transcriptional Phenotype of Dysfunctional Microglia in Neurodegenerative Diseases
|Immunity|2017|3k
Multiorgan and Renal Tropism of SARS-CoV-2
|New England Journal of Medicine|2020|1.9k
Classification of sporadic Creutzfeldt-Jakob disease based on molecular and phenotypic analysis of 300 subjects
|Annals of Neurology|1999|1.4k
Destabilization of β-catenin by mutations in presenilin-1 potentiates neuronal apoptosis
|Nature|1998|517
Mice with Combined Gene Knock-Outs Reveal Essential and Partially Redundant Functions of Amyloid Precursor Protein Family Members
|Journal of Neuroscience|2000|487