Cellular senescence promotes cancer metastasis by enhancing soluble E-cadherin production
Koichiro Kawaguchi(Kyoto Institute of Technology), Masataka Sugimoto(National Center for Geriatrics and Gerontology), Kaori Komoda(National Center for Geriatrics and Gerontology), Ryuta Mikawa(National Center for Geriatrics and Gerontology), Azusa Asai(National Center for Geriatrics and Gerontology)
Cited by 38
Related Papers
Opposing effects of Ets and Id proteins on p16INK4a expression during cellular senescence
|Nature|2001|646
Senolysis by glutaminolysis inhibition ameliorates various age-associated disorders
|Science|2021|434
Physical and Functional Interactions of the Arf Tumor Suppressor Protein with Nucleophosmin/B23
|Molecular and Cellular Biology|2004|377
Senolytic vaccination improves normal and pathological age-related phenotypes and increases lifespan in progeroid mice
|Nature Aging|2021|277
Nucleolar Arf Tumor Suppressor Inhibits Ribosomal RNA Processing
|Molecular Cell|2003|276