Opposing effects of Ets and Id proteins on p16INK4a expression during cellular senescence
Naoko Ohtani(Osaka Metropolitan University), Eiji Hara(The University of Osaka), Masataka Sugimoto(National Center for Geriatrics and Gerontology), Zoë Zebedee(Cancer Research UK Manchester Institute), Yasuhiro Ohashi, Gordon Peters(Cancer Research UK), Andrew D Sharrocks(University of Manchester), Thomas J. G. Huot(The Honourable Society of Lincoln's Inn), Julie A. Stinson(University of Manchester)
Cited by 646
Related Papers
An Essential Role for Senescent Cells in Optimal Wound Healing through Secretion of PDGF-AA
|Developmental Cell|2014|2k
Exosomes maintain cellular homeostasis by excreting harmful DNA from cells
|Nature Communications|2017|748
Glycolytic Enzymes Can Modulate Cellular Life Span
|Cancer Research|2005|640
Senolysis by glutaminolysis inhibition ameliorates various age-associated disorders
|Science|2021|434
Physical and Functional Interactions of the Arf Tumor Suppressor Protein with Nucleophosmin/B23
|Molecular and Cellular Biology|2004|377