Elimination of endogenous high molecular weight FGF2 prevents pressure-overload-induced systolic dysfunction, linked to increased FGFR1 activity and NR1D1 expression
Navid Koleini(Johns Hopkins University), Elissavet Kardami(University of Manitoba), Ian Dixon(St. Boniface Hospital), Natalie M. Landry(St. Boniface Hospital), Peter A. Cattini(University of Manitoba), Davinder S. Jassal(St. Boniface Hospital), David Cheung(St. Boniface Hospital), Robert R. Fandrich(St. Boniface Hospital), Raghu S. Nagalingam(St. Boniface Hospital), Barbara E. Nickel(St. Boniface Hospital), Michael P. Czubryt(St. Boniface Hospital)
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