Elimination of endogenous high molecular weight FGF2 prevents pressure-overload-induced systolic dysfunction, linked to increased FGFR1 activity and NR1D1 expressionNavid Koleini, Elissavet Kardami, Ian Dixon et al.|Cell and Tissue Research|2021Cited by 10
Changes in gene expression caused by genetic elimination of high molecular weight FGF2 are associated with prevention of stress‐induced cardiac systolic dysfunctionElissavet Kardami, Peter A. Cattini, Navid Koleini et al.|The FASEB Journal|2020Cited by 2
Abstract 838: High Molecular Weight FGF2 Contributes to Pressure Overload Induced Systolic Dysfunction by a Mechanism Associated With Modulation of the NR1D1 Orphan Nuclear Receptor ExpressionNavid Koleini, Elissavet Kardami, Peter A. Cattini et al.|Circulation Research|2019Cited by 1